The critical level of cadmium in human renal cortex: A reevaluation
References (4)
- et al.
In vivo measurement of liver and kidney cadmium in workers exposed to this metal: Its significance with respect to cadmium in blood and urine
Environ. Res.
(1981) - et al.
Characterization of the proteinuria induced by prolonged oral administration of cadmium in female rats
Toxicology
(1981)
There are more references available in the full text version of this article.
Cited by (44)
Effects of sub-chronic Cd exposure on levels of copper, selenium, zinc, iron and other essential metals in rat renal cortex
2016, Toxicology ReportsCitation Excerpt :These nephrotoxic effects occur when circulating Cd that is bound to low molecular proteins or thiol compounds is filtered at the glomerulus and/or presented to the basolateral cell surface and then taken up by the epithelial cells of the proximal tubule (for reviews see [36,63,64]. Over time, Cd accumulates in the cells to reach levels of 150–200 μg/g wet weight (equivalent to 450–600 μg/g dry weight) that cause toxic injury [18,23,43,44]. While these general effects of Cd on the kidney have been well-characterized, the specific molecular mechanisms through which Cd produces these effects have yet to be fully elucidated.
Toxic Metals: Cadmium
2014, Encyclopedia of Food SafetyEpidemiological application of early markers of nephrotoxicity
1989, Toxicology Letters
Copyright © 1983 Published by Elsevier Ireland Ltd.