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Original article
Associations between persistent organic pollutants, type 2 diabetes, diabetic nephropathy and mortality
  1. Brian A Grice1,
  2. Robert G Nelson1,
  3. Desmond E Williams2,
  4. William C Knowler1,
  5. Clinton Mason1,
  6. Robert L Hanson1,
  7. Kai McKeever Bullard2,
  8. Meda E Pavkov2
  1. 1 National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Phoenix, Arizona, USA
  2. 2 Division for Diabetes Translation, Centers for Disease Control and Prevention, Atlanta, Georgia, USA
  1. Correspondence to Dr Meda E Pavkov, Division for DIabetes Translation, Centers for Disease Control and Prevention, 4770 Buford Hwy NE MS F75, Atlanta 30341, Georgia, USA; mpavkov{at}cdc.gov

Footnotes

  • Parts of this article were presented at the 72nd Scientific Sessions of the American Diabetes Association, June 8–12, 2012, Philadelphia, Pennsylvania.

  • Contributors BAG, MEP, CCM, RGN and DEW contributed to the study design, data analysis, data interpretation and manuscript preparation. BAG, MEP, RGN and KMB contributed to manuscript preparation. RLH and WCK contributed to the study design, data interpretation and manuscript preparation. MEP is the guarantor of this work and, as such, had full access to all the data in the study and takes responsibility for the integrity of the data and the accuracy of the data analysis.

  • Funding This work was supported by the Intramural Program of the National Institute of Diabetes and Digestive and Kidney Diseases and by an inter-agency agreement between the Centers for Disease Control and Prevention and the National Institute of Diabetes and Digestive and Kidney Diseases (094FED4907651).

  • Disclaimer The findings and conclusions in this report are those of the authors and do not necessarily represent the official position of the National Institutes of Health or the Centers for Disease Control and Prevention.

  • Competing interests None declared.

  • Patient consent Each participant gave informed consent or assent.

  • Provenance and peer review Not commissioned; externally peer reviewed.

  • Correction notice This paper has been updated since it first published online. In table 1 the median age for the control group has been corrected to 21.0 (15.2-37.3).

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Footnotes

  • Parts of this article were presented at the 72nd Scientific Sessions of the American Diabetes Association, June 8–12, 2012, Philadelphia, Pennsylvania.

  • Contributors BAG, MEP, CCM, RGN and DEW contributed to the study design, data analysis, data interpretation and manuscript preparation. BAG, MEP, RGN and KMB contributed to manuscript preparation. RLH and WCK contributed to the study design, data interpretation and manuscript preparation. MEP is the guarantor of this work and, as such, had full access to all the data in the study and takes responsibility for the integrity of the data and the accuracy of the data analysis.

  • Funding This work was supported by the Intramural Program of the National Institute of Diabetes and Digestive and Kidney Diseases and by an inter-agency agreement between the Centers for Disease Control and Prevention and the National Institute of Diabetes and Digestive and Kidney Diseases (094FED4907651).

  • Disclaimer The findings and conclusions in this report are those of the authors and do not necessarily represent the official position of the National Institutes of Health or the Centers for Disease Control and Prevention.

  • Competing interests None declared.

  • Patient consent Each participant gave informed consent or assent.

  • Provenance and peer review Not commissioned; externally peer reviewed.

  • Correction notice This paper has been updated since it first published online. In table 1 the median age for the control group has been corrected to 21.0 (15.2-37.3).

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