Abstract
The aim of the second phase of the Pollution Atmosphérique et Affections Respiratoires Chroniques (PAARC) study, started in 1974, was to compare the long-term mortality between populations living in areas with different air pollution levels. In Bordeaux (France), four different areas were concerned by the study. The black smoke measures were realized between 1974 and 1981. After 1981, the stations set specifically for the study were not used any more. The purpose of this study was to estimate the evolution of air pollution in those areas between 1982 and 1997 using the measures of 12 Association de Prévention de la Pollution Atmosphérique (APPA) stations located in Bordeaux city but not in the PAARC areas. The method used was divided in three phases: a correlation study between the stations of the different networks, a selection of the pertinent stations and the setting up of indicators using the arithmetic means method. Monthly means concentrations were estimated from January 1982 to December 1997. Models showed a decrease in black smoke levels whatever the area. The difference in level from one area to another, existing between the areas in 1974, was still with predicted values in 1997, but less important. Black smoke mean concentration for 1982–1997 was, respectively, 16.4 and 16.2 μg/m3, in areas 1 and 2. It was a little bit higher in area 3 with 18.9 μg/m3. Area 4 still has the highest level with 26.3 μg/m3. To conclude, this method enabled to assess different air pollution levels at different times in the four areas of the PAARC study in Bordeaux. Those levels could be used to study the impact of the air pollution on long-term mortality on populations living in the areas considered.
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Acknowledgements
Supported by the Primequal-Predit program from the French Environment Ministry. The authors thank the APPA Bordeaux-Aquitaine committee for its contribution to the measurements.
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FILLEUL, L., BALDI, I., QUENEL, P. et al. Long-term air pollution indicator assessment: Example of black smoke in Bordeaux, France. J Expo Sci Environ Epidemiol 12, 226–231 (2002). https://doi.org/10.1038/sj.jea.7500222
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DOI: https://doi.org/10.1038/sj.jea.7500222