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We recently reported1 on the risk of incident asthma associated with traffic-related air pollution in a high-risk birth cohort in Vancouver, British Columbia. We have now examined the effect of two common polymorphisms in the glutathione-s-transferase P1 (GSTP1) gene that is known to metabolise oxidative species and which has been shown previously to modify risk of allergic disease associated with nitrogen dioxide (NO2) exposure.2
At 7 years of age, 173 children were assessed for asthma by paediatric allergists and peripheral blood was obtained from these children and their parents. DNA was genotyped by the Illumina BeadArray system (Illumina, San Diego, California, USA) following the …
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