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Substantial evidence suggests that the risk of lung cancer is magnified among individuals exposed to both tobacco smoke and asbestos. Previous studies have given inconsistent estimates of the strength of this interaction, possibly because of variation in several time related factors, such as cohort age, duration of follow up, and time since exposure, but findings reported by Reid et al from a study of Australian crocidolite miners and millers may help shed light on this question.1 Among this mature cohort of 2935 workers who were last exposed to asbestos over 30 years ago, on average, 18% had never smoked and 40% had quit. With adjustment for asbestos exposure, the risk of lung cancer declined steeply with time since ceasing smoking. The relative effect of asbestos appeared to be greater among non-smokers relative to smokers, consistent with greater-than-additive interaction. Scientific and practical implications of the research are discussed in an accompanying
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